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It has a long lasting effect and is essentially used in cases of high blood pressure (hypertension). In chronic stable angina pectoris and Prinzmetal's angina (vasospastic angina pectoris), it is used to treat and prevent attacks. Norvasc relaxes the vascular smooth muscle, dilating the blood arteries. Its detailed mechanism of action is not yet structured. The drug reduces ischemia because the arterioles dilate the peripheral vascular system making the myocardial function stable, because of which it consumes less energy and less oxygen. The dilated coronary artery in normal and ischemic regions causes a greater amount of oxygen to be released in the myocardium in patients with angina and variably reduces the action of vasoconstrictors in patients who smoke. Norvasc entirely metabolizes all inactive metabolites which are excreted through urine.

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